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Schaller Biology Lab

Summary:

"We are interested in the mechanisms by which plants grow and respond to changes in their environment. Plants make use of a diverse group of signaling compounds to regulate growth and development. Although some of these compounds were identified almost a century ago, only recently has significant progress been made in identifying the proteins involved in sensing and transducing these signals. Much of this work has been accomplished by using the plant Arabidopsis, which serves as a model organism for addressing basic questions in plant biology. My laboratory uses a combination of biochemical, molecular, and genetic strategies to analyze signaling pathways in Arabidopsis."

Research projects:
1. Mechanism of cytokinin signal transduction in Arabadopsis
2. Mechanism of ethylene signal transduction in Arabadopsis

People:

Resources:

Organisms and Viruses

  • ARR1 loss of function mutants ( Arabidopsis thaliana )

    Type-B mutants involving single and higher order loss-of-function mutations of ARR1, transcription factor/two-component response regulator.

  • ARR10 loss of function mutants ( Arabidopsis thaliana )

    Type-B mutants involving single and higher order loss-of-function mutations of ARR10, transcription factor/two-component response regulator.

  • ARR11 loss of function mutants ( Arabidopsis thaliana )

    Type-B mutants involving single and higher order loss-of-function mutations of ARR11, transcription factor/two-component response regulator.

  • ARR12 loss of function mutants ( Arabidopsis thaliana )

    Type-B mutants involving single and higher order loss-of-function mutations of ARR12, transcription factor/two-component response regulator.

  • ARR2 loss of function mutants ( Arabidopsis thaliana )

    Type-B mutants involving single and higher order loss-of-function mutations of ARR2, transcription factor/two-component response regulator.

  • EIN4 loss of function mutants ( Arabidopsis thaliana )

    Mutants involving single and higher order loss-of-function mutations of EIN4, ethylene binding/glycogen synthase kinase 3/protein histidine kinase/receptor.

  • ERS1 loss of function mutants ( Arabidopsis thaliana )

    Mutants involving single and higher order loss-of-function mutations of ERS1, ethylene binding/glycogen synthase kinase 3/protein histidine kinase/receptor.

  • ERS2 loss of function mutants ( Arabidopsis thaliana )

    Mutants involving single and higher order loss-of-function mutations of ERS2, ethylene binding/glycogen synthase kinase 3/protein histidine kinase/receptor.

  • ETR1 loss of function mutants ( Arabidopsis thaliana )

    Ethylene receptor mutants involving single and higher order loss-of-function mutations of ETR1.

  • ETR2 loss of function mutants ( Arabidopsis thaliana )

    Mutants involving single and higher order loss-of-function mutations of ETR2, ethylene binding/glycogen synthase kinase 3/protein histidine kinase/receptor.


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Last updated: 2011-11-28T12:02:14.207-05:00

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The eagle-i Consortium is supported by NIH Grant #5U24RR029825-02 / Copyright 2016